通过动物实验．观察AC960对微管蛋白聚合—解聚活性的影响。结果显示：AC960能明显抑制微管蛋白聚合，其作用略弱于秋水仙碱，且同时有抑制微管蛋白解聚作用。说明AC960抗癌机理可能在于影响微管蛋白的聚会—解聚活性，破坏纺缍丝形成，从而抑制癌细胞有丝分裂，促进其凋亡。 Through animal experiments. The effect of AC960 on the tubulin polymerization-depolymerization activity was observed. The results showed that: AC960 can significantly inhibit tubulin polymerization, its role is slightly weaker than colchicine, and at the same time inhibit the depolymerization of tubulin. It is suggested that the anti-cancer mechanism of AC960 may be affecting the convergence of tubulin-depolymerization activity, destroying the formation of spun silk, thereby inhibiting cancer cell mitosis and promoting its apoptosis.