肾小管性酸中毒(RTA)是一组以肾脏酸化功能缺陷为特征的高氯血症性代谢性酸中毒。即使内源性酸产量(即由膳食、代谢和消化道中盐基消耗而生成的酸)并未增多,单由肾脏分泌质子,也不能使血浆重碳酸盐保持于正常浓度。如表83一2所示:肾脏氢 Renal tubular acidosis (RTA) is a group of hyperchlorinated metabolic acidosis characterized by defects in renal acidification. Even with no increase in endogenous acid production (ie acid produced by diet, metabolism, and consumption of base in the digestive tract), the secretion of protons by the kidneys alone does not maintain plasma bicarbonate at normal concentrations. As shown in Table 83-2: Kidney Hydrogen