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肾小管性酸中毒(Renal tubular acidosis,RTA)系由远端肾小管分泌氢离子和/或近端肾小管对碳酸氢盐再吸收发生障碍,或二者共同存在而引起的持续性高氯性代谢性酸中毒伴各种电解质紊乱而无肾小球功能异常。RTA是可治之症,但若病人得不到早期诊治,可发展到不可逆性肾功能衰竭,1981年我院共收治4例,本文结合实践,重点分析与探讨RTA的诊断和治疗体会。例1:女,31岁,住院号428144,1981年8月18日入院。患者1976年左髋关节疼痛随即口渴、多饮、多尿,夜尿大于2900ml。外院拟诊为尿崩症。1978年4月以来全身骨痛,双肩、胸肋部、双髋、左膝、双踝关节均疼痛并乏力。体重
Renal tubular acidosis (RTA) is a persistent high chloride induced by the secretion of hydrogen ions from the distal tubule and / or the proximal tubule that is obstructive to bicarbonate reabsorption, or both Metabolic acidosis with a variety of electrolyte disorders without glomerular dysfunction. RTA is a treatable disease, but if the patient can not get early diagnosis and treatment, can develop to irreversible renal failure, in our hospital in 1981 were treated in 4 cases, this article with practice, focusing on analysis and discussion of RTA diagnosis and treatment experience. Example 1: Female, 31 years old, hospital number 428144, admitted on August 18, 1981. Patients with left hip pain in 1976 followed by thirst, polydipsia, polyuria, nocturia more than 2900ml. External hospital diagnosed as diabetes insipidus. Since April 1978 whole body pain, shoulders, chest ribs, double hip, left knee, both ankle joint pain and fatigue. body weight