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发生风湿性心脏病以后,共心脏所以能够维持在代偿阶段,主要是依靠心脏胀大(包括心脏肥厚或扩大)、窦性心动过速以及心循环血量和静脉回心血量的增加。在代偿功能不全阶段(心力衰竭),心脏更胀大,在负担相同的工作条件下,相对地此正常心脏耗氧更多,效率更低。这时心脏的进一步胀大,乃是心脏内剩余血量增多和心肌纤维更肥厚拉长的结果。洋地黄类药物只有提高心肌纤维收缩的力量和效率,排出剩余的
After the occurrence of rheumatic heart disease, the heart can maintain a compensatory phase, mainly relying on the expansion of the heart (including hypertrophy or enlargement), sinus tachycardia, and cardiopulmonary circulation and blood back to the amount of blood. In decompensated phase (heart failure), the heart is more swollen and under the same working conditions, the normal heart is relatively oxygen-consuming and less effective. At this moment, the further expansion of the heart is the result of an increase in the remaining blood in the heart and a more hypertrophic myocardial fiber. Digitalis drugs only improve the strength and efficiency of cardiac muscle contraction, the discharge of the remaining