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玉米幼苗在缺钙初期叶肉细胞能维持较高的ＳＯＤ活性，膜脂过氧化作用较弱；随缺钙时间延长，ＳＯＤ活性下降，尤其是Ｃｕ，Ｚｎ－ＳＯＤ活性急剧下降，膜脂过氧化作用加强，细胞器破坏．缺钙培养的叶片细胞超微结构的变化首先表现在叶绿体类囊体解体，随后质膜、线粒体膜、核膜和内质网膜等内膜系统紊乱和伤害，而膜系统的伤害可能是缺钙条件下受ＳＯＤ控制的膜脂过氧化作用增强的结果
In the early stage of calcium deficiency, the maize seedlings could maintain higher SOD activity and the peroxidation of membrane lipid was weak. With the prolongation of calcium deficiency, the activity of SOD decreased, especially the activity of Cu and Zn-SOD decreased sharply, and the membrane lipid peroxidation Strengthen organelle damage. Changes in the ultrastructure of leaf cells under calcium deficiency were first manifested in the disintegration of chloroplast thylakoids, followed by disorganization and injury of the endometrium, such as the plasma membrane, mitochondrial membrane, nuclear membrane and endoplasmic reticulum membrane, and injury to the membrane system Increased membrane lipid peroxidation under the control of SOD under calcium conditions