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编辑同志: 近年来众多文献认为慢性阻塞性肺疾病(COPD)患者高浓度吸氧致动脉血二氧化碳分压(PaCO_2)升高是由通气/血流(V/Q)比例失调加重所致。即低氧血症纠正后,缺氧性肺血管痉挛解除,血管扩张,肺内高(V/Q)区血流向低(V/Q)区。基本证据是生理死腔容积/潮气量(V_D/V_T)增大,进而认为是V_D增大(刘志光,等.高浓度氧疗致慢性阻塞性肺疾病患者动脉血二氧化碳分压增高的机理,中
Editor comrades: In recent years, many literatures consider the elevated concentration of oxygen in the patients with chronic obstructive pulmonary disease (COPD) leading to an increase in arterial blood pressure of carbon dioxide (PaCO 2) due to the increased imbalance of ventilation / blood flow (V / Q). That hypoxemia corrected, hypoxic pulmonary vasospasm, vasodilation, intrapulmonary high (V / Q) blood flow to the low (V / Q) zone. The basic evidence is that the volume of physiological dead space / tidal volume (V_D / V_T) increases, which is considered to be increased V_D (Liu Zhiguang, et al. Mechanism of elevated partial pressure of arterial carbon dioxide in patients with chronic obstructive pulmonary disease