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【目的】对中大304、MR1400、T1006和大区50等4个抗源进行稻纹枯病抗性遗传分析,为利用这些资源进行基因定位与育种提供依据。【方法】用感稻纹枯病的粳稻品种中野1211作母本,4个籼稻抗源品种中大304、MR1400、T1006和大区50分别作父本,组配4个杂交组合,获得F1和F2群体。用广西稻纹枯病菌优势致病型菌株AG-I-IA、GX-2以牙签嵌入法对F1和F2进行接种鉴定和遗传分析。【结果】抗源中大304、MR1400、T1006和大区50均表现中抗纹枯病,中野1211高感纹枯病;4个杂种F1植株对纹枯病的抗病等级为4.0～8.0级,表现为中抗～高感,平均为5.1～6.1级,为中感,群体抗性呈连续性分布;F2群体纹枯病抗性分离谱较广,抗性等级变化范围为2.0～9.0(R～HS),群体抗性呈连续单峰分布。抗源中大304、MR1400、T1006和大区50对水稻纹枯病的抗性遗传率分别为15.4%、31.1%、47.7%和43.8%。【结论】4个杂交组合的F1群体对纹枯病的抗性均介于其双亲之间,抗源中大304、MR1400、T1006和大区50对水稻纹枯病的抗性均为数量性状,受微效多基因控制。
【Objective】 Genetic analysis of rice sheath blight resistance was carried out on 4 resistance sources of Zhongda 304, MR1400, T1006 and Z50 in order to provide basis for gene mapping and breeding using these resources. 【Method】 The japonica rice variety Zhongye 1211 with rice sheath blight was used as female parent and four indica rice varieties Zhong-Nong 304, MR1400, T1006 and Zu-50 were used as male parents, F2 population. Inoculation and genetic analysis of F1 and F2 were carried out with toothpicks by using the dominant pathogenic strains AG-I-IA and GX-2 of rice sheath blight of Guangxi. 【Result】 The results showed that resistance to Rhizoctonia cerealis and resistance to Rhizoctonia solani in Zhongyuan 1211 and resistance to Rhizoctonia solani in four hybrid F1 plants were 4.0 ~ 8.0 , Showing moderate resistance to high susceptibility, with an average of 5.1 to 6.1, with a moderate flu resistance. The resistance of the population was continuously distributed. The spectrum of resistance to sheath blight of F2 population was broad, and the resistance range varied from 2.0 to 9.0 R ~ HS), the population resistance showed a continuous unimodal distribution. The resistance heritability of Zhongyuan 304, MR1400, T1006 and Z50 to rice sheath blight were 15.4%, 31.1%, 47.7% and 43.8%, respectively. 【Conclusion】 The resistance of F1 population to sheath blight in 4 cross combinations was between their parents. The resistances to resistance to sheath blight were resistance traits in Zhongyuan 304, MR1400, T1006 and Z50, , Controlled by the microscopic polygene.