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A 62-year-old male with decompensated liver cirrhosis due to hepatitis C virus developed severe hepatic encephaiopathy with status epileptic us. The blood ammonia level on admission was more than twice the normal level. Brain computed tomography and magnetic resonance imaging were normal. In addition, electroencephalogram showed diffuse sharp waves, consistent with hepatic encephaiopathy. The status epilepticus was resolved after antiepileptic therapy (phenytoin sodium) and treatment for hepatic encephaiopathy (Branched chain amino acids). The blood ammonia level normalized with the clinical improvement and the patient did not have a recurrence of status epilepticus after the end of the antiepileptic treatment. Additionally, the electroencephalogram showed normal findings. Thus, we diagnosed the patient as hepatic encephaiopathy with status epilepticus. We consider the status epilepticus of this patient to a rare and interesting finding in hepatic encephaiopathy.
A 62-year-old male with decompensated liver cirrhosis due to hepatitis C virus developed severe hepatic encephaiopathy with status epileptic us. The blood ammonia level on admission was more than twice the normal level. Brain computed tomography and magnetic resonance imaging were normal. In addition, electroencephalogram showed diffuse sharp waves, consistent with hepatic encephaiopathy. The status epilepticus was resolved after antiepileptic therapy (phenytoin sodium) and treatment for hepatic encephaiopathy (Branched chain amino acids). The blood ammonia level normalized with the clinical improvement and the patient did not, a recurrence of status epilepticus after the end of the antiepileptic treatment..,, we diagnosed the patient as hepatic encephaiopathy with status epilepticus. We consider the status epilepticus of this patient to a rare and interesting finding. in hepatic encephaiopathy.