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肝硬化失代偿期的钾代谢紊乱,主要为低钾血症。但高钾血症亦非罕见,且可危及病人生命,我科自1985年以来,共收治肝硬化失代偿期患者108例,并低钾血症11例(10.18%),无1例死亡。而并发高钾血症6例(5.55%),有2例死亡。笔者认为应引起高度重视。现对其发病机理与处理进行初步探讨。例1:男,64岁,主因腹泻、腹胀4个月伴鼻衄于1988年7月8日入院。体检:黄疸、腹水、脾大,化验肝功能异常,确诊为肝硬化失代偿期,除给保肝治疗外,每天口服安体舒通80mg,间断腹腔内注射速尿,多巴胺,腹水明显减少,曾一度出
Disorders of liver cirrhosis potassium metabolism disorder, mainly hypokalemia. However, hyperkalemia is not uncommon and can endanger the life of patients. Since 1985, our department has treated 108 patients with decompensated cirrhosis and 11 patients (10.18%) with hypokalemia, none of whom died . In 6 cases of hyperkalemia (5.55%), 2 died. I think it should attract great attention. Now its pathogenesis and treatment of a preliminary study. Example 1: Male, 64 years old, mainly due to diarrhea, bloating for 4 months with nasal obstruction on July 8, 1988 admission. Physical examination: jaundice, ascites, splenomegaly, laboratory liver function abnormalities, diagnosed as decompensated liver cirrhosis, in addition to the liver treatment, spironolactone 80mg daily, intermittent intraperitoneal injection furosemide, dopamine, ascites decreased significantly , Once out