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口腔扁平苔藓(oral lichen planus OLP)是一种口腔黏膜的慢性炎症,此病与口腔癌前病变有关[1]。尽管病变机制尚不清楚,随着基因研究的发展和多聚酶链技术的应用。研究显示:错配修复基因(hMSH2)、p63基因、人乳头状瘤病毒(HPV)、爱泼斯坦-巴尔病毒(EBV)等在OLP癌变机制中发挥一定的作用。
Oral lichen planus (OLP) is a chronic inflammation of the oral mucosa that is associated with premalignant oral lesions [1]. Although the pathological mechanism is not clear, with the development of genetic research and the application of polymerase chain technology. Studies have shown that mismatch repair gene (hMSH2), p63 gene, human papilloma virus (HPV), Epstein-Barr virus (EBV) play a role in the carcinogenesis of OLP.