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近年来发现 ,核因子 κB(NF κB)活化是介导脑缺血 /再灌注 (I/R)后炎性损伤的中心环节 ,但临床尚缺乏有效地调控I/R后脑内NF κB活化的措施 ;既往研究发现 ,FNS可下调脑出血大鼠脑内NF κB的蛋白表达 ,但目前尚不清楚FNS能否直接调控I/R后脑内NF κB的活化 ,临床经皮电刺激小
In recent years, it has been found that the activation of nuclear factor κB (NF κB) is the central part of inflammatory injury induced by cerebral ischemia / reperfusion (I / R). However, the lack of effective regulation of NF κB activation in the brain after I / R Previous studies have found that FNS can down-regulate the expression of NF-κB in the rat brain after intracerebral hemorrhage. However, it is not clear whether FNS can directly regulate the activation of NF-κB in the brain after I / R.